- What is kwashiorkor? What are its symptoms and remedies?
- What is merasmus? What are its symptoms and remedies?
- What is the full form of P.E.M.? Explain it.
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Kwashiorkor is an acute form of childhood protein-energy malnutrition characterized by edema, irritability, anorexia, ulcerating dermatoses, and an enlarged liver with fatty infiltrates. The presence of edema caused by poor nutrition defines kwashiorkor.
The defining sign of kwashiorkor in a malnourished child is pedal edema (swelling of the feet). Other signs include a distended abdomen, an enlarged liver with fatty infiltrates, thinning hair, loss of teeth, skin depigmentation anddermatitis. Children with kwashiorkor often develop irritability and anorexia.
Treatment of kwashiorkor depends upon its severity. Fluid and electrolyte imbalances may need to be corrected with intravenous fluids, and infections may require treatment with Antibiotics.
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2.Marasmus is a form of severe protein-energy malnutrition characterized by energy deficiency.
A child with marasmus looks emaciated. Body weight may be reduced to less than 80% of the average weight that corresponds to the height
The malnutrition associated with marasmus leads to extensive tissue and muscle wasting, as well as variable edema. Other common characteristics include dry skin, loose skin folds hanging over the buttocks (glutei) and armpit (axillae), etc. There is also drastic loss of adipose tissue (body fat) from normal areas of fat deposits like buttocks and thighs. The afflicted are often fretful, irritable, and voraciously hungry.
It is necessary to treat not only the symptoms but also the complications of the disorder, including infections, dehydration, and circulation disorders, which are frequently lethal and lead to high mortality if ignored.
Ultimately, marasmus can progress to the point of no return when the body's machinery for protein synthesis, itself made of protein, has been degraded to the point that it cannot handle any protein. At this point, attempts to correct the disorder by giving food or protein are futile.
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PEM is also referred to as protein-energy malnutrition. It develops in children and adults whose consumption of protein and energy (measured by calories) is insufficient to satisfy the body's nutritional needs. While pure protein deficiency can occur when a person's diet provides enough energy but lacks the protein minimum, in most cases the deficiency will be dual. PEM may also occur in persons who are unable to absorb vital nutrients or convert them to energy essential for healthy tissue formation and organ function.
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Kwashiorkor
Kwashiorkor is an acute form of childhood protein-energy malnutrition characterized by edema, irritability, anorexia, ulcerating dermatoses, and an enlarged liver with fatty infiltrates. The presence of edema caused by poor nutrition defines kwashiorkor.
The defining sign of kwashiorkor in a malnourished child is pedal edema (swelling of the feet). Other signs include a distended abdomen, an enlarged liver with fatty infiltrates, thinning hair, loss of teeth, skin depigmentation anddermatitis. Children with kwashiorkor often develop irritability and anorexia.
Treatment of kwashiorkor depends upon its severity. Fluid and electrolyte imbalances may need to be corrected with intravenous fluids, and infections may require treatment with Antibiotics.
A child with marasmus looks emaciated. Body weight may be reduced to less than 80% of the average weight that corresponds to the height The malnutrition associated with marasmus leads to extensive tissue and muscle wasting, as well as variable edema. Other common characteristics include dry skin, loose skin folds hanging over the buttocks (glutei) and armpit (axillae), etc. There is also drastic loss of adipose tissue (body fat) from normal areas of fat deposits like buttocks and thighs. The afflicted are often fretful, irritable, and voraciously hungry. It is necessary to treat not only the symptoms but also the complications of the disorder, including infections, dehydration, and circulation disorders, which are frequently lethal and lead to high mortality if ignored.
Ultimately, marasmus can progress to the point of no return when the body's machinery for protein synthesis, itself made of protein, has been degraded to the point that it cannot handle any protein. At this point, attempts to correct the disorder by giving food or protein are futile.
PEM
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